Biology of Ticks, Volume 2This is the second of a two-volume work on the biology, morphology, ecology, disease relationships, and control of ticks. Volume 2 explores survival strategies of non-nidicolous ticks (those dispersed throughout the open landscape and attacking passing hosts) versus nidicolous ticks (those surviving in caves, burrows, nests, or man-made shelters). It also examines immunological responses to tick parasitism, the role of ticks in disease transmission, and the control of ticks through acaricides and recent innovative approaches using knowledge of tick and host ecology, tick pheromones, hormones, and modelling. An appendix is also included, with details on methods for collecting ticks in the natural environment, preparing ticks for study, and laboratory rearing. This book is a worthy complement to the first volume's outstanding achievement, and will be of interest to entomologists, physicians, veterinarians, and public health officers. |
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Page 137
... host cell membrane , forming a recess that eventually closes into a vacuole ( Fig . 26.16 ) . Sporozoite invasion is an active process in which both the living sporozoite and host cell participates . Receptor sites on the membrane of ...
... host cell membrane , forming a recess that eventually closes into a vacuole ( Fig . 26.16 ) . Sporozoite invasion is an active process in which both the living sporozoite and host cell participates . Receptor sites on the membrane of ...
Page 138
... host cell , secretions from the rhoptries and microspheres dissolve the enclosing vacuolar membrane and allow the escape of the parasite into the host cell cytoplasm ( Shaw et al . , 1991 ) , in contrast with the malarial parasites ...
... host cell , secretions from the rhoptries and microspheres dissolve the enclosing vacuolar membrane and allow the escape of the parasite into the host cell cytoplasm ( Shaw et al . , 1991 ) , in contrast with the malarial parasites ...
Page 144
... host cell is deteriorating ; the cytoplasm has largely disappeared and the cell membrane is also breaking down . Hcm = Host cell membrane ( remnant ) ; Mt mitochondria ; Nu = nucleus ( of host cell ) ; Rh = rhoptries . Photograph credit ...
... host cell is deteriorating ; the cytoplasm has largely disappeared and the cell membrane is also breaking down . Hcm = Host cell membrane ( remnant ) ; Mt mitochondria ; Nu = nucleus ( of host cell ) ; Rh = rhoptries . Photograph credit ...
Contents
ECOLOGY OF NONNIDOCOLOUS TICKS | 3 |
ECOLOGY OF NIDICOLOUS TICKS | 66 |
ACQUIRED IMMUNITY TO TICKS | 92 |
Copyright | |
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abundance acaricide adult ticks Africa Amblyomma americanum andersoni animals antibodies antigens appendiculatus areas Argas argasid attack Babesia babesiosis birds Borrelia Burgdorfer burgdorferi burrows cattle caused clinical cycle deer Dermacentor develop diagnosis diapause dogs Ecology engorged enzootic Epidemiology erythrocytes females Figure forest genus habitat heartwater host cell host-seeking humidity immune important incubation isolated Ixodes dammini ixodid laboratory larvae livestock Lyme borreliosis Lyme disease mammals membrane merozoites midgut molting nests nidicolous Norval numbers nymphal nymphs occur organisms Ornithodoros oviposition paralysis parasites pathogen period permission proteins questing rabbits region relapsing fever reported Rhipicephalus ricinus rickettsiae RMSF rodents salivary glands schizonts seasonal activity sheep small mammals Sonenshine southern Spielman spirochetes sporozoites spotted fever stages survival symptoms temperature Theileria tick control tick feeding tick paralysis tick population tick species tick vectors tick-borne tick-borne diseases tissues transmission transmitted transovarial transstadial treatment tularemia unfed vaccine variabilis vector tick vegetation vertebrate virus viruses white-footed mice wild