Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
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Page 156
... symptoms , and reactivate quiescent dis- ease.21 These potentiating effects may be mediated by breaking the epithelial barrier or initiating a mucosal inflammatory response . Following epidemics of Shi- gella , Salmonella , or Yersinia ...
... symptoms , and reactivate quiescent dis- ease.21 These potentiating effects may be mediated by breaking the epithelial barrier or initiating a mucosal inflammatory response . Following epidemics of Shi- gella , Salmonella , or Yersinia ...
Page 358
... symptoms . The mechanisms underlying the symptoms of IBD are no better understood than are the etiologies . However , in- creased insights into human pathophysiology offer clues to the genesis of certain clinical features and today ...
... symptoms . The mechanisms underlying the symptoms of IBD are no better understood than are the etiologies . However , in- creased insights into human pathophysiology offer clues to the genesis of certain clinical features and today ...
Page 699
... symptoms or signs suggestive of Crohn's disease , eight ultimately needed pouch excision or proximal diversion . Of the remaining 15 patients who lacked suspicious symptoms , only one had lost his pouch with an average follow - up ...
... symptoms or signs suggestive of Crohn's disease , eight ultimately needed pouch excision or proximal diversion . Of the remaining 15 patients who lacked suspicious symptoms , only one had lost his pouch with an average follow - up ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis