Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 55
... patients in whom Her- mansky - Pudlak syndrome and IBD occur to- gether ( 231 , 246 , 247 ) . Clearly , conclusions about the association of this syndrome with IBD cannot be made on the basis of such mea- ger information . As with ...
... patients in whom Her- mansky - Pudlak syndrome and IBD occur to- gether ( 231 , 246 , 247 ) . Clearly , conclusions about the association of this syndrome with IBD cannot be made on the basis of such mea- ger information . As with ...
Page 305
... IBD patients and also in their unaffected family members . Table 13.7 presents a sum- mary of the diseases most commonly associ- ated with IBD . Table 13.6 Genetic Syndromes With Inflammatory Bowel Disease Turner's syndrome Hermansky ...
... IBD patients and also in their unaffected family members . Table 13.7 presents a sum- mary of the diseases most commonly associ- ated with IBD . Table 13.6 Genetic Syndromes With Inflammatory Bowel Disease Turner's syndrome Hermansky ...
Page 485
... IBD patients to infections . Since most cases of bacterial endocarditis associated with IBD involve organisms commonly found in the gas- trointestinal tract ( 97 ) , the question has been raised of whether all IBD patients should re ...
... IBD patients to infections . Since most cases of bacterial endocarditis associated with IBD involve organisms commonly found in the gas- trointestinal tract ( 97 ) , the question has been raised of whether all IBD patients should re ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually