Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 14
... body in a manner that will cause its tissues to react with increased in- tensity to subsequent contact with the infective agent . " More than 100 years elapsed before the important role of the gastrointestinal tract in the immune ...
... body in a manner that will cause its tissues to react with increased in- tensity to subsequent contact with the infective agent . " More than 100 years elapsed before the important role of the gastrointestinal tract in the immune ...
Page 151
... bodies to enterobacterial common antigen ( ECA ) and to E. coli serotype 014 have been identified in ulcerative colitis ... body in the sera of UC patients but not Crohn's or other colitis patients . The ligand for this antibody is a 40 ...
... bodies to enterobacterial common antigen ( ECA ) and to E. coli serotype 014 have been identified in ulcerative colitis ... body in the sera of UC patients but not Crohn's or other colitis patients . The ligand for this antibody is a 40 ...
Page 504
... body 40.4 ( 32.7 ) 38.1 ( 10 ) 41.5 ( 11 ) .1220 Pain or suffering 39.5 ( 28.5 ) 34.3 ( 14 ) 42.0 ( 10 ) .0001b Feeling out of control 38.0 ( 32.6 ) 37.7 ( 11 ) 38.2 ( 13 ) .8295 Attractiveness 35.8 ( 32.0 ) 32.1 ( 16 ) 37.6 ( 14 ) ...
... body 40.4 ( 32.7 ) 38.1 ( 10 ) 41.5 ( 11 ) .1220 Pain or suffering 39.5 ( 28.5 ) 34.3 ( 14 ) 42.0 ( 10 ) .0001b Feeling out of control 38.0 ( 32.6 ) 37.7 ( 11 ) 38.2 ( 13 ) .8295 Attractiveness 35.8 ( 32.0 ) 32.1 ( 16 ) 37.6 ( 14 ) ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually