Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 948
... calcium oxalate or urate itself further enhances the tendency to stone formation . Treatment of patients with intestinal disease and uric acid renal stones has been discussed in detail ( 261-266 ) . Briefly , any recovered stone should ...
... calcium oxalate or urate itself further enhances the tendency to stone formation . Treatment of patients with intestinal disease and uric acid renal stones has been discussed in detail ( 261-266 ) . Briefly , any recovered stone should ...
Page 950
... calcium . Large supplements of calcium may increase the amount of calcium excreted in the urine . Unless oxalate absorption and urinary oxalate excretion is " normalized ” by the calcium treat- ment , the rise in urinary calcium from ...
... calcium . Large supplements of calcium may increase the amount of calcium excreted in the urine . Unless oxalate absorption and urinary oxalate excretion is " normalized ” by the calcium treat- ment , the rise in urinary calcium from ...
Page 951
... calcium pump of plasma membrane . In Peterlick M , Bronner F ( eds ) : Mole- cular and Cellular Regulators of Calcium and Phosphate Metabolism . New York , John Wiley & Sons , 1990 . 44. Favus MJ , Rothplia SC , Coe FL : Kinetic ...
... calcium pump of plasma membrane . In Peterlick M , Bronner F ( eds ) : Mole- cular and Cellular Regulators of Calcium and Phosphate Metabolism . New York , John Wiley & Sons , 1990 . 44. Favus MJ , Rothplia SC , Coe FL : Kinetic ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually