Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 195
... collagen in the gut wall ( 29 ) . Traditionally , it was thought that col- lagen was synthesized by fibroblasts , but more recent work has demonstrated the ability of en- teric smooth muscle cells to synthesize the pro- tein ( 33 ) and ...
... collagen in the gut wall ( 29 ) . Traditionally , it was thought that col- lagen was synthesized by fibroblasts , but more recent work has demonstrated the ability of en- teric smooth muscle cells to synthesize the pro- tein ( 33 ) and ...
Page 196
... collagen synthesis ( panel A ) , noncollagen protein ( panel B ) , and in collagen relative to total protein synthesis ( panel C ) . The action of TGF - B ( shown by square symbols ) was selective in that similar responses were not ...
... collagen synthesis ( panel A ) , noncollagen protein ( panel B ) , and in collagen relative to total protein synthesis ( panel C ) . The action of TGF - B ( shown by square symbols ) was selective in that similar responses were not ...
Page 543
... collagen band immediately beneath the luminal epithe- lium . In the presence of a colitis , all patients with a collagen band this thick ( about 15 μ ) will have symptoms . However , unless specifically looked for , this is easily ...
... collagen band immediately beneath the luminal epithe- lium . In the presence of a colitis , all patients with a collagen band this thick ( about 15 μ ) will have symptoms . However , unless specifically looked for , this is easily ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually