Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 131
... crypt abscess in a patient with active IBD . Numerous neu- trophils have transmigrated across the crypt epithelium and have collected within the crypt lumen . Such events raise several issues concerning neutrophil - epithelial inter ...
... crypt abscess in a patient with active IBD . Numerous neu- trophils have transmigrated across the crypt epithelium and have collected within the crypt lumen . Such events raise several issues concerning neutrophil - epithelial inter ...
Page 519
... crypt abscesses and a chronic com- ponent involving the lamina propria more diffusely . Aggregates of neutrophils near and invading the crypt epithelium form crypt ab- scesses that , when numerous , is a characteris- tic and reliable ...
... crypt abscesses and a chronic com- ponent involving the lamina propria more diffusely . Aggregates of neutrophils near and invading the crypt epithelium form crypt ab- scesses that , when numerous , is a characteris- tic and reliable ...
Page 567
... crypt architecture , not present in acute self - limited colitis . Crypt ar- chitecture was considered distorted if there were two or more branched crypts in the well- oriented central core of an adequate size biopsy . Increased numbers ...
... crypt architecture , not present in acute self - limited colitis . Crypt ar- chitecture was considered distorted if there were two or more branched crypts in the well- oriented central core of an adequate size biopsy . Increased numbers ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually