Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 584
... Esophagus , Stomach , and Duodenum The esophagus , stomach , and duodenum can be examined satisfactorily by either the single contrast or double contrast technique . If single contrast examination is elected , fluoroscopy with careful ...
... Esophagus , Stomach , and Duodenum The esophagus , stomach , and duodenum can be examined satisfactorily by either the single contrast or double contrast technique . If single contrast examination is elected , fluoroscopy with careful ...
Page 613
... Esophagus , Stomach , and Duodenum Involvement of the stomach and duodenum is uncommon , and disease of the esophagus is rare . Either single or double contrast meth- ods can be used to identify the pathologic changes . Early roentgen ...
... Esophagus , Stomach , and Duodenum Involvement of the stomach and duodenum is uncommon , and disease of the esophagus is rare . Either single or double contrast meth- ods can be used to identify the pathologic changes . Early roentgen ...
Page 629
... esophagus with narrowing and deep ulceration . apparent . These changes are not specific , how- ever , and may be found in ischemia , infection , and other diseases . With healing and fibrosis in Crohn's disease , the bowel wall ...
... esophagus with narrowing and deep ulceration . apparent . These changes are not specific , how- ever , and may be found in ischemia , infection , and other diseases . With healing and fibrosis in Crohn's disease , the bowel wall ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually