Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 482
... fluid loss also may promote stone formation . Strategies to decrease enteric hyperoxaluria should begin with reducing ox- alate intake , limiting dietary fat , substituting fats with medium - chain triglycerides , increasing fluid ...
... fluid loss also may promote stone formation . Strategies to decrease enteric hyperoxaluria should begin with reducing ox- alate intake , limiting dietary fat , substituting fats with medium - chain triglycerides , increasing fluid ...
Page 742
... fluid are required to provide the daily energy requirements . When fluid restric- tion is necessary , 20 % lipid emulsions , which have twice the caloric density of most TPN so- lutions , can be used . In addition , 50 % dex- trose can ...
... fluid are required to provide the daily energy requirements . When fluid restric- tion is necessary , 20 % lipid emulsions , which have twice the caloric density of most TPN so- lutions , can be used . In addition , 50 % dex- trose can ...
Page 743
... fluids per day . It reabsorbs most of this volume in the small bowel . The amount reabsorbed in the small intestine depends on the nature of the meal . With a meat and salad meal that is low in osmolality , most of the fluid is absorbed ...
... fluids per day . It reabsorbs most of this volume in the small bowel . The amount reabsorbed in the small intestine depends on the nature of the meal . With a meat and salad meal that is low in osmolality , most of the fluid is absorbed ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually