Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 301
... genetic syndromes that feature IBD , and associations between IBD and genetic markers . The principal thesis of this chapter is that the inflammatory bowel diseases are funda- mentally genetic diseases with complex non- mendelian ...
... genetic syndromes that feature IBD , and associations between IBD and genetic markers . The principal thesis of this chapter is that the inflammatory bowel diseases are funda- mentally genetic diseases with complex non- mendelian ...
Page 317
... genetic or more genetic determinis- tic ; UC may be more stochastic and thus may be more immunologic Some genetic aspects of Crohn's disease and UC are distinct Environmental factors are likely not microenvironmental , but ...
... genetic or more genetic determinis- tic ; UC may be more stochastic and thus may be more immunologic Some genetic aspects of Crohn's disease and UC are distinct Environmental factors are likely not microenvironmental , but ...
Page 324
... genetic predispositions . More precise empiric risks to family members essential for genetic counseling and genetic analysis are being developed . Significant progress is being made in elucidating the na- ture of the genetic ...
... genetic predispositions . More precise empiric risks to family members essential for genetic counseling and genetic analysis are being developed . Significant progress is being made in elucidating the na- ture of the genetic ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually