Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 76
... increased in chronic ( 5 weeks ) but not acute ( 5 days ) or healed coli- tis ( 37 ) . In addition , the increased PGE , pro- duction does not appear to be responsible for the IL - 2 alterations . In the mouse model , immunohistochemi ...
... increased in chronic ( 5 weeks ) but not acute ( 5 days ) or healed coli- tis ( 37 ) . In addition , the increased PGE , pro- duction does not appear to be responsible for the IL - 2 alterations . In the mouse model , immunohistochemi ...
Page 224
... Increased titers of antibodies to cow's milk proteins such as ca- sein and ß - lactoglobulin have been reported in patients with ulcerative colitis ( 281 ) and pe- diatric Crohn's disease patients ( 284 ) , but oth- ers have found them ...
... Increased titers of antibodies to cow's milk proteins such as ca- sein and ß - lactoglobulin have been reported in patients with ulcerative colitis ( 281 ) and pe- diatric Crohn's disease patients ( 284 ) , but oth- ers have found them ...
Page 948
... increased to produce a urine volume of 1500 to 2000 mL / day . A persistently acid urine pH can be corrected by oral administration of a potas- sium / sodium citrate mixture such as Polycitra or Urocit - K . Urine pH should be increased ...
... increased to produce a urine volume of 1500 to 2000 mL / day . A persistently acid urine pH can be corrected by oral administration of a potas- sium / sodium citrate mixture such as Polycitra or Urocit - K . Urine pH should be increased ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually