Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 186
... inflammatory mediators and cytokines that regulate the inflammatory response but rather the chronology of inflammation . The ... process . Far less work has gone into defining the process by which inflammation is shut down under normal ...
... inflammatory mediators and cytokines that regulate the inflammatory response but rather the chronology of inflammation . The ... process . Far less work has gone into defining the process by which inflammation is shut down under normal ...
Page 199
... inflammatory process . That is , they respond to the presence of in- flammatory cells and their mediators by alter- ing their normal physiologic functions , but do not themselves contribute to the ongoing in- flammation . This view ...
... inflammatory process . That is , they respond to the presence of in- flammatory cells and their mediators by alter- ing their normal physiologic functions , but do not themselves contribute to the ongoing in- flammation . This view ...
Page 588
... Inflammatory Process Beginning with the rectum , continuity of the inflammatory process often is cited as charac- teristic of CUC , and this distinction frequently is correct . If the patient is examined soon after the onset of symptoms ...
... Inflammatory Process Beginning with the rectum , continuity of the inflammatory process often is cited as charac- teristic of CUC , and this distinction frequently is correct . If the patient is examined soon after the onset of symptoms ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually