Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 419
... intakes of sodium , chloride , and fluid are adequate , patients with ileostomies do not become depleted ; however , negative sodium balance may follow periods of diminished oral intake , vomiting , or excessive losses in perspira- tion ...
... intakes of sodium , chloride , and fluid are adequate , patients with ileostomies do not become depleted ; however , negative sodium balance may follow periods of diminished oral intake , vomiting , or excessive losses in perspira- tion ...
Page 739
... intake . Often they may replace rather than supple- ment energy intake from food . Patients who develop early satiety and cannot increase their total energy intake require tube - feeding or parenteral nutrition . Enteral Feeding of ...
... intake . Often they may replace rather than supple- ment energy intake from food . Patients who develop early satiety and cannot increase their total energy intake require tube - feeding or parenteral nutrition . Enteral Feeding of ...
Page 745
... intake of small volumes of a glucose - electrolyte solution . The composition of this solution is glucose 200 mmol / L with sodium 85 to 90 mmol / L , potas- sium 12 mmol / L , bicarbonate 9 mmol / L , and chloride 80 to 90 mmol / L ...
... intake of small volumes of a glucose - electrolyte solution . The composition of this solution is glucose 200 mmol / L with sodium 85 to 90 mmol / L , potas- sium 12 mmol / L , bicarbonate 9 mmol / L , and chloride 80 to 90 mmol / L ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually