Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 206
... Lamina Propria Lymphocytes ( LPLs ) The intestinal lamina propria contains an abundance of B cells , plasma cells , T cells , and macrophages , as well as a lesser number of other cells such as eosinophils and mast cells ( 7 ) . The ...
... Lamina Propria Lymphocytes ( LPLs ) The intestinal lamina propria contains an abundance of B cells , plasma cells , T cells , and macrophages , as well as a lesser number of other cells such as eosinophils and mast cells ( 7 ) . The ...
Page 215
... Lamina Propria Cells Cells isolated from lamina propria include T cells , B cells , and macrophages ( 44 ) ; the av- erage recoveries of these different cell types are shown in Table 10.3 . Data on lamina pro- pria cell isolates have ...
... Lamina Propria Cells Cells isolated from lamina propria include T cells , B cells , and macrophages ( 44 ) ; the av- erage recoveries of these different cell types are shown in Table 10.3 . Data on lamina pro- pria cell isolates have ...
Page 216
... lamina propria cells . Lamina propria cells both from control and IBD patients show evidence of in vivo activa- tion . In addition to increased expression of activation markers and spontaneous immuno- globulin production , lamina ...
... lamina propria cells . Lamina propria cells both from control and IBD patients show evidence of in vivo activa- tion . In addition to increased expression of activation markers and spontaneous immuno- globulin production , lamina ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually