Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 182
... macrophages with various ef- fector capabilities ( 31 , 32 ) . There are many classes of tissue macrophages that express dif- ferent surface antigens and have different functional capabilities ( proliferation , phagocy- tosis , tumor ...
... macrophages with various ef- fector capabilities ( 31 , 32 ) . There are many classes of tissue macrophages that express dif- ferent surface antigens and have different functional capabilities ( proliferation , phagocy- tosis , tumor ...
Page 227
... MACROPHAGES Macrophages form a significant proportion of the cells present in both the noninflamed and inflamed intestinal lamina propria . Histo- chemical and immunohistochemical stains indicate that intestinal macrophages are het ...
... MACROPHAGES Macrophages form a significant proportion of the cells present in both the noninflamed and inflamed intestinal lamina propria . Histo- chemical and immunohistochemical stains indicate that intestinal macrophages are het ...
Page 271
... Macrophages Neutrophils Fibroblasts Smooth muscle cell Functional Effect ↓ Proliferation ↓ Cytolytic activity Promotes chemotaxis cyte , and macrophage colonies ; granulocyte- macrophage CSF ( GM - CSF ) for granulocytes and macrophages ...
... Macrophages Neutrophils Fibroblasts Smooth muscle cell Functional Effect ↓ Proliferation ↓ Cytolytic activity Promotes chemotaxis cyte , and macrophage colonies ; granulocyte- macrophage CSF ( GM - CSF ) for granulocytes and macrophages ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually