Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 193
... muscle contrac- tion ( 16 ) . In contrast , as shown in Figure 9.3 , nematode - induced colitis is associated with a decrease in muscle contractility ( 17 ) . In addi- tion , it is evident that circular and longitudinal muscle respond ...
... muscle contrac- tion ( 16 ) . In contrast , as shown in Figure 9.3 , nematode - induced colitis is associated with a decrease in muscle contractility ( 17 ) . In addi- tion , it is evident that circular and longitudinal muscle respond ...
Page 194
... muscle from the small intestine of patients with Crohn's dis- ease , there was no increase in the maximum tension generated but the sensitivity of the tis- sue to carbachol was significantly increased ( Fig . 9.6 ) . These data confirm ...
... muscle from the small intestine of patients with Crohn's dis- ease , there was no increase in the maximum tension generated but the sensitivity of the tis- sue to carbachol was significantly increased ( Fig . 9.6 ) . These data confirm ...
Page 195
... MUSCLE CELLS IN STRICTURE FORMATION IN INFLAMMATORY BOWEL DISEASE In ulcerative colitis , strictures usually are re- versible and probably reflect changes in muscle bulk , whereas in Crohn's disease , strictures also reflect the ...
... MUSCLE CELLS IN STRICTURE FORMATION IN INFLAMMATORY BOWEL DISEASE In ulcerative colitis , strictures usually are re- versible and probably reflect changes in muscle bulk , whereas in Crohn's disease , strictures also reflect the ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually