Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 131
... neutrophil - epithelial inter- actions in IBD . How do neutrophils transmigrate ? How do they move across epithelia ? What functional consequence does this have to the epithelium ? How might neutrophils in the crypt lumen influence ...
... neutrophil - epithelial inter- actions in IBD . How do neutrophils transmigrate ? How do they move across epithelia ? What functional consequence does this have to the epithelium ? How might neutrophils in the crypt lumen influence ...
Page 182
... neutrophil activation is the phagocytosis and killing of bacteria . In inflammatory bowel disease huge numbers of neutrophils migrate into the in- flamed intestine without the phagocytosis of bacteria . The stimulus for neutrophil ...
... neutrophil activation is the phagocytosis and killing of bacteria . In inflammatory bowel disease huge numbers of neutrophils migrate into the in- flamed intestine without the phagocytosis of bacteria . The stimulus for neutrophil ...
Page 228
... neutrophils are produced per day . The neutrophil's phagocytic activity can be separated into multi- ple distinct ... neutrophils are not . The infiltration of the intestinal lesions of IBD by neutrophils is the hallmark of active IBD ...
... neutrophils are produced per day . The neutrophil's phagocytic activity can be separated into multi- ple distinct ... neutrophils are not . The infiltration of the intestinal lesions of IBD by neutrophils is the hallmark of active IBD ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually