Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 101
... pathogenesis should target viruses known to stimulate an autoimmune re- sponse , arthritogenic Yersinia and Chlamydia strains , and epithelial proteins that display ho- mology with enteric pathogens and commen- sal bacteria . ROLE OF ...
... pathogenesis should target viruses known to stimulate an autoimmune re- sponse , arthritogenic Yersinia and Chlamydia strains , and epithelial proteins that display ho- mology with enteric pathogens and commen- sal bacteria . ROLE OF ...
Page 110
... pathogenesis of intestinal and systemic inflammation . Com- mensal bacteria could induce chronic idio- pathic IBD by a number of mechanisms , as listed in Table 4.6 . The two primary hypothe- ses are an appropriate immunologic response ...
... pathogenesis of intestinal and systemic inflammation . Com- mensal bacteria could induce chronic idio- pathic IBD by a number of mechanisms , as listed in Table 4.6 . The two primary hypothe- ses are an appropriate immunologic response ...
Page 118
... pathogenesis of IBD . The key mediators of immunoregulation need to be determined and should be explored using environmentally relevant antigens and stimulants such as bacte- rial cell wall polymers , antigens of commensal bacteria ...
... pathogenesis of IBD . The key mediators of immunoregulation need to be determined and should be explored using environmentally relevant antigens and stimulants such as bacte- rial cell wall polymers , antigens of commensal bacteria ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually