Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 37
... proctitis ( 16 , 77 , 102 ) . A Balti- more survey indicated that less than 10 % of the proctitis population is ever hospitalized , and the proportions never hospitalized are similar for Scandinavian and other countries . Thus most of ...
... proctitis ( 16 , 77 , 102 ) . A Balti- more survey indicated that less than 10 % of the proctitis population is ever hospitalized , and the proportions never hospitalized are similar for Scandinavian and other countries . Thus most of ...
Page 963
... proctitis were not corre- lated with responsiveness to corticosteroids or 5 - ASA compounds . Because of the apparent tendency of proctitis to extend proximally among teenagers and young adults , we are re- luctant to forecast a ...
... proctitis were not corre- lated with responsiveness to corticosteroids or 5 - ASA compounds . Because of the apparent tendency of proctitis to extend proximally among teenagers and young adults , we are re- luctant to forecast a ...
Page 964
... proctitis ) , nev- ertheless is an aggravating factor . Research in progress indicates a close relationship between the central nervous system and the enteric nervous system of the gut . As more informa- tion on this relationship and ...
... proctitis ) , nev- ertheless is an aggravating factor . Research in progress indicates a close relationship between the central nervous system and the enteric nervous system of the gut . As more informa- tion on this relationship and ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually