Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 117
... Response Tissue Response Viral or bacterial infections , PG - PS LPS FMLP NSAIDs , Bacterial & toxins dietary antigens Downregulation Resolution , tissue repair Healing , no residual damage normal host Acute inflammation Genetically ...
... Response Tissue Response Viral or bacterial infections , PG - PS LPS FMLP NSAIDs , Bacterial & toxins dietary antigens Downregulation Resolution , tissue repair Healing , no residual damage normal host Acute inflammation Genetically ...
Page 186
... response but rather the chronology of inflammation . The influx of neutrophils into the lamina propria in shigellosis occurs over a few days , whereas the influx of neutrophils in ulcerative colitis goes on for weeks to months . In ...
... response but rather the chronology of inflammation . The influx of neutrophils into the lamina propria in shigellosis occurs over a few days , whereas the influx of neutrophils in ulcerative colitis goes on for weeks to months . In ...
Page 286
... response of those cells in vitro ( Table 12.1 ) . Together , the effects that have been identified suggest that these neuropeptides have a variety of impor- tant immunoregulatory functions that are di- rectly relevant to the intestine ...
... response of those cells in vitro ( Table 12.1 ) . Together , the effects that have been identified suggest that these neuropeptides have a variety of impor- tant immunoregulatory functions that are di- rectly relevant to the intestine ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually