Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 337
Joseph B. Kirsner, Roy Gerrard Shorter. and severity of colonic involvement . Fever , prostration , dehydration , and postural hy- potension accompany the most severe disease . The abdomen may be protruberant as a result of colonic atony ...
Joseph B. Kirsner, Roy Gerrard Shorter. and severity of colonic involvement . Fever , prostration , dehydration , and postural hy- potension accompany the most severe disease . The abdomen may be protruberant as a result of colonic atony ...
Page 338
... severe symp- toms of ulcerative colitis to recognize dilata- tion of the colon and incipient or established perforation . Approximately 10 % of patients with severe attacks of colitis demonstrate colonic dilatation ( diameter of the ...
... severe symp- toms of ulcerative colitis to recognize dilata- tion of the colon and incipient or established perforation . Approximately 10 % of patients with severe attacks of colitis demonstrate colonic dilatation ( diameter of the ...
Page 339
... Severe 40 30. are typical of reported population - based studies ( 23 ) . The outcome of the first attack is greatly in- fluenced by the extent of colonic involvement , the severity of the attack , and the age of the patient . The extent ...
... Severe 40 30. are typical of reported population - based studies ( 23 ) . The outcome of the first attack is greatly in- fluenced by the extent of colonic involvement , the severity of the attack , and the age of the patient . The extent ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually