Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 651
... steroid with both enhanced biodegra- dation and increased potency compared with the traditional steroids as a result of modifica- tions at the 17 - alpha position similar to triam- cinolone , but with an acetyl rather than an acetonide ...
... steroid with both enhanced biodegra- dation and increased potency compared with the traditional steroids as a result of modifica- tions at the 17 - alpha position similar to triam- cinolone , but with an acetyl rather than an acetonide ...
Page 652
... steroid - induced osteo- porosis while fluoride salts and biphospho- nates are being evaluated ( 174 ) . Estrogen should be considered in postmenopausal women on steroids ( 173 ) . A serious complication of steroids in chil- dren is ...
... steroid - induced osteo- porosis while fluoride salts and biphospho- nates are being evaluated ( 174 ) . Estrogen should be considered in postmenopausal women on steroids ( 173 ) . A serious complication of steroids in chil- dren is ...
Page 686
... steroids in patients referred for steroid dependency . These individuals will require continued high- dose mesalamine ( usually combining oral and topical therapy ) to maintain remission once steroids have been successfully tapered . In ...
... steroids in patients referred for steroid dependency . These individuals will require continued high- dose mesalamine ( usually combining oral and topical therapy ) to maintain remission once steroids have been successfully tapered . In ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually