Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 142
... stomach provides a pH incompatible with survival of most bacteria and also provides an environment optimal for the activity of pepsin . Postgastrectomy patients are more likely to be colonized with potentially invasive pathogens ...
... stomach provides a pH incompatible with survival of most bacteria and also provides an environment optimal for the activity of pepsin . Postgastrectomy patients are more likely to be colonized with potentially invasive pathogens ...
Page 584
... stomach empties , the radiologist carries out fluoroscopy with vigorous palpation of the opacified bowel with the patient in various po- sitions at frequent intervals until the entire bowel has been visualized . A film is made after ...
... stomach empties , the radiologist carries out fluoroscopy with vigorous palpation of the opacified bowel with the patient in various po- sitions at frequent intervals until the entire bowel has been visualized . A film is made after ...
Page 613
... Stomach , and Duodenum Involvement of the stomach and duodenum is uncommon , and disease of the esophagus is rare . Either single or double contrast meth- ods can be used to identify the pathologic changes . Early roentgen changes in ...
... Stomach , and Duodenum Involvement of the stomach and duodenum is uncommon , and disease of the esophagus is rare . Either single or double contrast meth- ods can be used to identify the pathologic changes . Early roentgen changes in ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually