Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 307
... STUDIES Genetic marker studies are one approach to identification of the actual genes that pre- dispose to IBD . The specific methods include population - association studies and family - link- age studies . In association studies ...
... STUDIES Genetic marker studies are one approach to identification of the actual genes that pre- dispose to IBD . The specific methods include population - association studies and family - link- age studies . In association studies ...
Page 311
... studies of complement in IBD patients and their families may be im- portant in determining the etiology of at least a subset of IBD . Another approach to the role of the com- plement system is searching for disease associa- tions with ...
... studies of complement in IBD patients and their families may be im- portant in determining the etiology of at least a subset of IBD . Another approach to the role of the com- plement system is searching for disease associa- tions with ...
Page 391
... studies on IBD to present analyses , relates to such medical advances as expanded diagnostic studies ( e.g. , endoscopy , double contrast barium studies , and computed tomography ) , the increased ca- pacity for more complete ...
... studies on IBD to present analyses , relates to such medical advances as expanded diagnostic studies ( e.g. , endoscopy , double contrast barium studies , and computed tomography ) , the increased ca- pacity for more complete ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually