Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 670
... sulfasalazine at equal doses of 5 - ASA , results were similar for olsalazine ( 97-100 ) . The primary advantage of olsalazine is in the maintenance of remission of ulcerative colitis where the compound is better tolerated than ...
... sulfasalazine at equal doses of 5 - ASA , results were similar for olsalazine ( 97-100 ) . The primary advantage of olsalazine is in the maintenance of remission of ulcerative colitis where the compound is better tolerated than ...
Page 695
... SULFASALAZINE Single - Agent Treatment of Active Disease Sulfasalazine is commonly prescribed for the treatment of active Crohn's disease . Its use is supported by several small clinical reports , as well as by controlled trials . Among ...
... SULFASALAZINE Single - Agent Treatment of Active Disease Sulfasalazine is commonly prescribed for the treatment of active Crohn's disease . Its use is supported by several small clinical reports , as well as by controlled trials . Among ...
Page 696
... sulfasalazine and prednisone also was evaluated by the NCCDS ( 2 ) . The combination appeared even less effective than prednisone alone . The addi- tion of sulfasalazine to 6 - methylprednisolone in the ECCDS likewise offered no ...
... sulfasalazine and prednisone also was evaluated by the NCCDS ( 2 ) . The combination appeared even less effective than prednisone alone . The addi- tion of sulfasalazine to 6 - methylprednisolone in the ECCDS likewise offered no ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually