Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 19
... cerative colitis and Crohn's disease with genet- ically mediated conditions , such as ankylosing spondylitis and Turner's syndrome for ulcera- tive colitis , and psoriasis and the Hermansky- Pudlak syndrome for Crohn's disease , added ...
... cerative colitis and Crohn's disease with genet- ically mediated conditions , such as ankylosing spondylitis and Turner's syndrome for ulcera- tive colitis , and psoriasis and the Hermansky- Pudlak syndrome for Crohn's disease , added ...
Page 537
... colitis differ from ulcerative colitis ( Fig . 25.14 , A , B , and C ) . In infectious colitis , the architecture is preserved ; the ac- tive inflammation tends to be superficial with epithelial destruction , in contrast to ulcera- tive ...
... colitis differ from ulcerative colitis ( Fig . 25.14 , A , B , and C ) . In infectious colitis , the architecture is preserved ; the ac- tive inflammation tends to be superficial with epithelial destruction , in contrast to ulcera- tive ...
Page 564
... Crohn's colitis originates over the M cell in the epithelium overlying submucosal lymphatic follicles . The inflamma- tion subsequently advances to the superficial layers . The earliest tissue response in ulcera- tive colitis is an ...
... Crohn's colitis originates over the M cell in the epithelium overlying submucosal lymphatic follicles . The inflamma- tion subsequently advances to the superficial layers . The earliest tissue response in ulcera- tive colitis is an ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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Common terms and phrases
abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually