Inflammatory Bowel DiseaseJoseph B. Kirsner, Roy Gerrard Shorter This text is intended to help provide the practical perspectiv= e practitioners need to manage patients suffering from inflammatory bowel disease. This fourth edition contains 11 new chapters and is illustrated with photographs and detailed line drawings. |
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Page 345
... urinary bladder , or to an abscess cavity in the mesen- tery . Fistulas to the urinary bladder can mani- fest as a urinary tract infection , pyelonephritis , pneumaturia , or even fecaluria . With extensive and long - standing involve ...
... urinary bladder , or to an abscess cavity in the mesen- tery . Fistulas to the urinary bladder can mani- fest as a urinary tract infection , pyelonephritis , pneumaturia , or even fecaluria . With extensive and long - standing involve ...
Page 482
... urinary excretion of in- hibitors of calcium urolithiasis such as magne- sium , pyrophosphate , and citrate . Further- more , diminished urine volume secondary to excessive enteric fluid loss also may promote stone formation ...
... urinary excretion of in- hibitors of calcium urolithiasis such as magne- sium , pyrophosphate , and citrate . Further- more , diminished urine volume secondary to excessive enteric fluid loss also may promote stone formation ...
Page 948
... urinary pH almost always is low ( 5 to 5.5 ) , the volume is reduced , and the concentration of uric acid is increased . Because the pKa of uric acid is 5.42 , much of the urinary urate will be present as the protonated , poorly soluble ...
... urinary pH almost always is low ( 5 to 5.5 ) , the volume is reduced , and the concentration of uric acid is increased . Because the pKa of uric acid is 5.42 , much of the urinary urate will be present as the protonated , poorly soluble ...
Contents
Historical Antecedents of Inflammatory | 3 |
The Epidemiology of Idiopathic Inflammatory | 31 |
Naturally Occurring and Experimental Models | 71 |
Copyright | |
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abnormalities acid activity acute altered antibodies antigens appear areas associated bacterial biopsies blood cancer carcinoma cause cells changes chronic Clin clinical coli colon common compared complications controls Crohn's disease cytokines demonstrated develop diagnosis diarrhea dysplasia ease effects enteritis epithelial cells epithelium et al evidence examination expression factor Figure findings fistulas function Gastroenterol Gastroenterology gene genetic growth human identified ileum immune important incidence increased indicate induced infection inflammation inflammatory bowel disease initial intestinal involved lamina propria lesions less levels lymphocytes major mechanisms mediators mucosal neutrophils normal observed occur patients patients with Crohn's population possible present production protein rats recent receptor rectal rectum regional relatives reported response risk role severe similar small bowel specific strictures studies suggest symptoms Table therapy tients tion tissue tive treatment ulcerative colitis usually