The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
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Results 1-3 of 84
Page 40
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
Page 202
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
Page 276
... become the pleural cavities , the lining of the walls of the canals becomes the parietal pleura . The region where the visceral pleura reflects off the lungs and becomes continuous with the parietal pleura shifts medially and becomes ...
... become the pleural cavities , the lining of the walls of the canals becomes the parietal pleura . The region where the visceral pleura reflects off the lungs and becomes continuous with the parietal pleura shifts medially and becomes ...
Contents
foraminacontinued | 11 |
gamma globulins 261 | 21 |
mucous 23 24 36 53 113 136 | 113 |
Copyright | |
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Common terms and phrases
abdominal abnormalities abscess acute airway alveolar alveoli aorta asthma atelectasis basal biopsy blood body breathing bron bronchial bronchogenic capillary carcinoma cartilage cause caveola cavity cell chest wall chest x-ray film chronic CIBA clinical Continued cor pulmonale costal cough cysts diagnosis diaphragm diaphragmatic diffuse disease drainage drugs dyspnea edema elastic embolism emphysema esophagus fibers fibrosis flow fluid glands hemorrhage hilar hypoxemia increase infection infiltration intercostal interstitial involved lateral lesions ligation lower lobe lung volume lymph nodes medial mediastinal mediastinum membrane metastatic muscle nerve Netter CIBA Netter M.D. nodules normal obstruction occur oxygen parietal pleura patients peripheral pleural pneumonia pneumothorax posterior pressure pulmo pulmonary artery pulmonary embolism resection respiratory result right lung roentgenogram sarcoidosis SECTION IV PLATE segment sputum superior vena cava surface surgical symptoms syndrome therapy thoracic tion tissue trachea tracheobronchial treatment tube tuberculosis tumor upper lobe usually vascular vein vena cava venous ventilation vertebral vessels