The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
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Page 82
SECTION IV PLATE 121 Principles of Tests for Monitoring Anticoagulant Therapy
A. Prothrombin time ( PT ) ; more sensitive to warfarin effect Tissue
thromboplastin plus Therapeutic range Toxic level Normal Reagent Tests for
Monitoring ...
SECTION IV PLATE 121 Principles of Tests for Monitoring Anticoagulant Therapy
A. Prothrombin time ( PT ) ; more sensitive to warfarin effect Tissue
thromboplastin plus Therapeutic range Toxic level Normal Reagent Tests for
Monitoring ...
Page 169
( 1 ) C. B. 101 1.0 After cessation of smoking 8 Chronic Obstructive Pulmonary
Disease ( Continued ) Vmax . 50 0.8Vmax . l / sec 6 Cdyn Cstat Normal range 4 .
Vmax . 25 0.6While smoking Range after cessation of smoking 2 0.420 40 60 80
...
( 1 ) C. B. 101 1.0 After cessation of smoking 8 Chronic Obstructive Pulmonary
Disease ( Continued ) Vmax . 50 0.8Vmax . l / sec 6 Cdyn Cstat Normal range 4 .
Vmax . 25 0.6While smoking Range after cessation of smoking 2 0.420 40 60 80
...
Page 175
Even in normal subjects , flow rates over the lower three - fourths of the vital
capacity are limited by the mechanical properties of the lungs and airways rather
than by the amount of driving pressure the subject can generate . In contrast ,
peak ...
Even in normal subjects , flow rates over the lower three - fourths of the vital
capacity are limited by the mechanical properties of the lungs and airways rather
than by the amount of driving pressure the subject can generate . In contrast ,
peak ...
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Common terms and phrases
abnormalities acute airway alveolar alveoli amphotericin appear artery associated asthma become blood body branches breathing bronchial bronchus capillary carbon carcinoma cartilage cause cavity cells changes chest chronic CIBA clinical common complications contain Continued develop diagnosis diaphragm diffuse disease edema effect embolism emphysema examination fibers fibrosis flow fluid frequently function heart hyperventilation increased indicated infection inspiration intercostal internal interstitial involved lateral lesions less lobe lower lung lymph major mechanical mediastinal mediastinum membrane muscle nerve Netter nodes normal obstruction occur organisms oxygen patients pattern PLATE pleural pneumonia position posterior present pressure primary produce pulmonary rare resistance respiratory response result SECTION IV PLATE seen segment severe side skin space sputum superior surface symptoms syndrome therapy thoracic thoracotomy tion tissue trachea treatment tube tumors upper usually vein venous ventilation vessels volume wall x-ray