The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 89
Superficial thrombophlebitis in the legs or thighs rarely gives rise to emboli but
may signal a deep venous thrombosis . It is the loose propagating thrombus in
the deep veins that constitutes the hazard of pulmonary embolization . When
broken ...
Superficial thrombophlebitis in the legs or thighs rarely gives rise to emboli but
may signal a deep venous thrombosis . It is the loose propagating thrombus in
the deep veins that constitutes the hazard of pulmonary embolization . When
broken ...
Page 90
A large cuff is placed about the thigh and rapidly inflated to 50 mm Hg ; this
temporarily obstructs venous outflow . After the limb volume has stabilized , the
cuff is suddenly deflated and the maximal rate at which the calf volume empties ...
A large cuff is placed about the thigh and rapidly inflated to 50 mm Hg ; this
temporarily obstructs venous outflow . After the limb volume has stabilized , the
cuff is suddenly deflated and the maximal rate at which the calf volume empties ...
Page 226
Superficial thrombophlebitis in the legs or thighs rarely gives rise to emboli but
may signal a deep venous thrombosis . It is the loose propagating thrombus in
the deep veins that constitutes the hazard of pulmonary embolization . When
broken ...
Superficial thrombophlebitis in the legs or thighs rarely gives rise to emboli but
may signal a deep venous thrombosis . It is the loose propagating thrombus in
the deep veins that constitutes the hazard of pulmonary embolization . When
broken ...
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Common terms and phrases
abnormalities acute airway alveolar alveoli amphotericin appear artery associated asthma become blood body branches breathing bronchial bronchus capillary carbon carcinoma cartilage cause cavity cells changes chest chronic CIBA clinical common complications contain Continued develop diagnosis diaphragm diffuse disease edema effect embolism emphysema examination fibers fibrosis flow fluid frequently function heart hyperventilation increased indicated infection inspiration intercostal internal interstitial involved lateral lesions less lobe lower lung lymph major mechanical mediastinal mediastinum membrane muscle nerve Netter nodes normal obstruction occur organisms oxygen patients pattern PLATE pleural pneumonia position posterior present pressure primary produce pulmonary rare resistance respiratory response result SECTION IV PLATE seen segment severe side skin space sputum superior surface symptoms syndrome therapy thoracic thoracotomy tion tissue trachea treatment tube tumors upper usually vein venous ventilation vessels volume wall x-ray