Behavioral Neurology and Neuropsychology, Second EditionTodd E. Feinberg, Martha J. Farah The leading clinical reference on behavioral neurology! This state-of-the-art second edition reflects groundbreaking coverage of both clinical and theoretical aspects of brain-behavior studies. Features five new chapters in such rapidly expanding areas as cerebral plasticity, functional brain imaging, alterations in states of consciousness, and genetics of neural development. |
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Page 159
... cause anomic aphasia . If the lesions are large and extend into lateral temporal lobe or hemorrhage dissects up into the deep tempo- ral white matter , patients may present with Wernicke's aphasia or TCSA . Trauma can also cause large ...
... cause anomic aphasia . If the lesions are large and extend into lateral temporal lobe or hemorrhage dissects up into the deep tempo- ral white matter , patients may present with Wernicke's aphasia or TCSA . Trauma can also cause large ...
Page 349
... caused by peripheral lesions is much less likely to cause unawareness of deficit in the absence of significant dementia , while patients with occipital in- farctions may manifest unawareness without clouding of consciousness . Previous ...
... caused by peripheral lesions is much less likely to cause unawareness of deficit in the absence of significant dementia , while patients with occipital in- farctions may manifest unawareness without clouding of consciousness . Previous ...
Page 629
... cause , and about 2 percent of these patients had an infective cause . Moreover , increasing global importance of both AIDS dementia complex ( ADC ) and variant Creutzfeldt - Jakob disease make the con- sideration of an infective cause ...
... cause , and about 2 percent of these patients had an infective cause . Moreover , increasing global importance of both AIDS dementia complex ( ADC ) and variant Creutzfeldt - Jakob disease make the con- sideration of an infective cause ...
Contents
The Development of Modern Behavioral Neurology and Neuropsychology | 3 |
The Mental Status Exam | 23 |
Principles of Neuropsychological Assessment | 33 |
Copyright | |
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activity agnosia Alzheimer Alzheimer's disease amnesia amnesic anatomic Ann Neurol anosognosia aphasia apraxia Arch Neurol areas artery assessment associated atrophy attention auditory basal forebrain basal ganglia behavioral bilateral brain Broca's aphasia cerebral Clin clinical Cogn cognitive color confabulation correlates cortex cortical damage Damasio deficits diagnosis disorders dissociation dysfunction dyslexia effects epilepsy fMRI frontal lobe functional gyrus Heilman KM hippocampal human imaging impairment infarction injury involved language lesions Lewy bodies medial motor neglect neuroimaging Neurol Neurosurg Psychiatry Neurology Neuropsy neuropsychological Neurosci normal nucleus object parietal parietal lobe Parkinson's disease pathologic patients pattern percent performance phonologic posterior prefrontal processing progressive supranuclear palsy prosopagnosia Psychol recognition regions reported response retrieval right hemisphere seizures semantic memory sensory simultanagnosia somatosensory spatial specific speech stimuli stroke structures subcortical suggested symptoms syndrome tactile tasks temporal lobe tests thalamic tients tion vascular dementia verbal visual white matter word