The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 92
But the pylephlebitic abscesses may develop from purulent infections anywhere
in the portal system and subsequent multiple embolisms in the intrahepatic portal
vein branches . Inflammation of the bile ducts can lead to cholangitic abscesses ...
But the pylephlebitic abscesses may develop from purulent infections anywhere
in the portal system and subsequent multiple embolisms in the intrahepatic portal
vein branches . Inflammation of the bile ducts can lead to cholangitic abscesses ...
Page 102
DIRECT EXTENSION TO LUNG ABSCESS 8 . VASCULAR ROUTE TO LUNG
ABSCESS 9 . ... In the submucosa the amebae , living on tissue constituents and
red cells , produce small cavities ( not abscesses ) which eventually
communicate ...
DIRECT EXTENSION TO LUNG ABSCESS 8 . VASCULAR ROUTE TO LUNG
ABSCESS 9 . ... In the submucosa the amebae , living on tissue constituents and
red cells , produce small cavities ( not abscesses ) which eventually
communicate ...
Page 103
CUTANEOUS FISTULA BO SOS RELATIVELY SMALL ACTINOMYCOTIC
ABSCESS ACTINOMYCOSIS M VE 22 DO ... The typical initial localizations of the
abscesses are the jaws , the lung and the intestine , especially cecum and
appendix .
CUTANEOUS FISTULA BO SOS RELATIVELY SMALL ACTINOMYCOTIC
ABSCESS ACTINOMYCOSIS M VE 22 DO ... The typical initial localizations of the
abscesses are the jaws , the lung and the intestine , especially cecum and
appendix .
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall