The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 68
These connections carry vessels which become anastomoses . The liver cells in
those parts of the lobules that have been separated from their original
parenchymal environment also start to multiply , and this regenerative effort
results in cell ...
These connections carry vessels which become anastomoses . The liver cells in
those parts of the lobules that have been separated from their original
parenchymal environment also start to multiply , and this regenerative effort
results in cell ...
Page 69
When , in the course of the fibrotic process , the smaller branches of the portal
vein become compressed and in part even obliterated , the share of hepatic
blood coming from the artery increases relatively to the amount of blood from the
portal ...
When , in the course of the fibrotic process , the smaller branches of the portal
vein become compressed and in part even obliterated , the share of hepatic
blood coming from the artery increases relatively to the amount of blood from the
portal ...
Page 111
Bulging - outward subcapsular nodules may even become pedunculated . The
irregularity of the structural elements ( liver cell plates not arranged around a
central vein , proliferated bile ducts and connective tissue ) and the occasional ...
Bulging - outward subcapsular nodules may even become pedunculated . The
irregularity of the structural elements ( liver cell plates not arranged around a
central vein , proliferated bile ducts and connective tissue ) and the occasional ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall