The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 47
The resulting biliverdin - ironglobin product , called verdohemoglobin , loses its
iron and globin components and becomes biliverdin , which , subsequently , is
transformed by reduction into bilirubin . This pigment gives the red diazo reaction
...
The resulting biliverdin - ironglobin product , called verdohemoglobin , loses its
iron and globin components and becomes biliverdin , which , subsequently , is
transformed by reduction into bilirubin . This pigment gives the red diazo reaction
...
Page 48
Exaggerated red cell damage and hemolysis give rise to an excess of biliverdin
and , subsequently , of bilirubin which raises the indirect reacting bilirubin level in
the serum . In these cases the skin is of a reddish hue superimposed on the pale
...
Exaggerated red cell damage and hemolysis give rise to an excess of biliverdin
and , subsequently , of bilirubin which raises the indirect reacting bilirubin level in
the serum . In these cases the skin is of a reddish hue superimposed on the pale
...
Page 49
Direct and indirect reacting bilirubin may be increased ; the former because of
regurgitation of biliary substances , the latter probably urobilinogen excretion
varies because of the inconstant flow of inspissated bile from liver to duodenum .
Direct and indirect reacting bilirubin may be increased ; the former because of
regurgitation of biliary substances , the latter probably urobilinogen excretion
varies because of the inconstant flow of inspissated bile from liver to duodenum .
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall