The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 112
99 C sto BE GS 2008 Tumors II Primary Hepatic Carcinoma FORTES ho BADOS
ODOS OS HE he GA B ES DES RA o LE BORES l DEO ST 3 ESP treba OR BOD
80 pohon Se 1038 OE OOK PO OP EVENTOS NODULAR LO 32 RE RED an re ...
99 C sto BE GS 2008 Tumors II Primary Hepatic Carcinoma FORTES ho BADOS
ODOS OS HE he GA B ES DES RA o LE BORES l DEO ST 3 ESP treba OR BOD
80 pohon Se 1038 OE OOK PO OP EVENTOS NODULAR LO 32 RE RED an re ...
Page 113
Less frequent are other histologic varieties . The carcinoma cells may reveal
extensive fatty metamorphosis not present in the rest of the liver . Sometimes ,
large elongated multinuclear giant cells are present or even predominate the
picture .
Less frequent are other histologic varieties . The carcinoma cells may reveal
extensive fatty metamorphosis not present in the rest of the liver . Sometimes ,
large elongated multinuclear giant cells are present or even predominate the
picture .
Page 114
The biologic characteristics of carcinomas of the intrahepatic bile ducts differ
distinctly , however , from cholangiocellular carcinoma , which resembles
biologically hepatocellular carcinoma . It appears , therefore , justified to consider
...
The biologic characteristics of carcinomas of the intrahepatic bile ducts differ
distinctly , however , from cholangiocellular carcinoma , which resembles
biologically hepatocellular carcinoma . It appears , therefore , justified to consider
...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall