The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 42
SOLO 1933 os DES SES SED Cholesterol belongs to the steroids and occurs in
the body either as free alcohol or as fatty acid ester . The liver forms cholesterol
from small hydrocarbon compounds ; it esterifies free cholesterol and stores both
...
SOLO 1933 os DES SES SED Cholesterol belongs to the steroids and occurs in
the body either as free alcohol or as fatty acid ester . The liver forms cholesterol
from small hydrocarbon compounds ; it esterifies free cholesterol and stores both
...
Page 50
... CHOLESTEROL ESTERS RATIO ESTIMATION OF DEGREE OF LIVER
DAMAGE CHOLESTEROL ESTERS I RATIO CHOLESTEROL RATIO
CHOLESTEROL NPN ELEVATED BILIRUBIN ELEVATED HIPPURIC ACID
SYNTHESIS NORMAL ...
... CHOLESTEROL ESTERS RATIO ESTIMATION OF DEGREE OF LIVER
DAMAGE CHOLESTEROL ESTERS I RATIO CHOLESTEROL RATIO
CHOLESTEROL NPN ELEVATED BILIRUBIN ELEVATED HIPPURIC ACID
SYNTHESIS NORMAL ...
Page 129
The specks consist of cholesterol dissolved in fat within histiocytes , which give a
bright red color in Sudan stains . These histiocytes accumulate in groups within
the mucosal folds of the gallbladder , which lose their regularity and normal ...
The specks consist of cholesterol dissolved in fat within histiocytes , which give a
bright red color in Sudan stains . These histiocytes accumulate in groups within
the mucosal folds of the gallbladder , which lose their regularity and normal ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall