The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tractCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 40
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
Page 77
Frank Henry Netter. LIPOGENIC FACTORS 3 . MICROCALCULI IN DILATED BILE DUCTULES 1 . CENTRILOBULAR. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT PROTEIN DECREASED METABOLIC NEEDS LIPOTROPIC FACTORS Starvation ...
Frank Henry Netter. LIPOGENIC FACTORS 3 . MICROCALCULI IN DILATED BILE DUCTULES 1 . CENTRILOBULAR. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT PROTEIN DECREASED METABOLIC NEEDS LIPOTROPIC FACTORS Starvation ...
Page 79
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
Common terms and phrases
abdominal abnormal abscesses acid amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. CIBA nodes normal OCIBA organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall