The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tractCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 43
... increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if they are osteoblas- tic ...
... increased osteo- blastic activity . It is very high in such bone diseases as ricketts , osteomalacia and Paget's disease . It is moderately elevated with most carcinoma metastases to the bone , especially so if they are osteoblas- tic ...
Page 48
... Increased production or decreased excretion of biliary pigments leads to jaundice without bile flow impairment . Hemo- lytic jaundice , sometimes more , sometimes less pronounced , is an example of the overproduction of bile pigment and ...
... Increased production or decreased excretion of biliary pigments leads to jaundice without bile flow impairment . Hemo- lytic jaundice , sometimes more , sometimes less pronounced , is an example of the overproduction of bile pigment and ...
Page 74
... increased and reabsorption of tissue fluid is im- paired . If hypoproteinemia complicates portal hypertension , a rapid development of ascites may be expected and occurs promptly , e.g. , after hemorrhage from ruptured esophageal ...
... increased and reabsorption of tissue fluid is im- paired . If hypoproteinemia complicates portal hypertension , a rapid development of ascites may be expected and occurs promptly , e.g. , after hemorrhage from ruptured esophageal ...
Common terms and phrases
abdominal abnormal abscesses acid amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. CIBA nodes normal OCIBA organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall