The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 7
The fat droplets or vacuoles in normal liver cells do not exceed 4 microns in
diameter . ... The fat droplets become gradually larger until the liver cell
cytoplasm is studded with droplets of different size , the nucleus , however , still
remaining in ...
The fat droplets or vacuoles in normal liver cells do not exceed 4 microns in
diameter . ... The fat droplets become gradually larger until the liver cell
cytoplasm is studded with droplets of different size , the nucleus , however , still
remaining in ...
Page 49
JAUNDICE WITH IMPAIRMENT OF BILE FLOW > LIVER CELL DEGENERATION
COMPLETE EXTRAHEPATIC ... CHOLESTASIS HEPATITIS , CIRRHOSIS O SA
BILE CANALICULI LI DISTENDED LIVER CELLS DAMAGED DEDO BLOOD ...
JAUNDICE WITH IMPAIRMENT OF BILE FLOW > LIVER CELL DEGENERATION
COMPLETE EXTRAHEPATIC ... CHOLESTASIS HEPATITIS , CIRRHOSIS O SA
BILE CANALICULI LI DISTENDED LIVER CELLS DAMAGED DEDO BLOOD ...
Page 63
SECTION XVII – PLATE 4 FEATURES OF HEPATIC REGENERATION AND
ATROPHY Cells of the liver plates constantly disappear and are replaced by new
cells by either mitotic or amitotic division . Binucleate cells are , therefore , seen ...
SECTION XVII – PLATE 4 FEATURES OF HEPATIC REGENERATION AND
ATROPHY Cells of the liver plates constantly disappear and are replaced by new
cells by either mitotic or amitotic division . Binucleate cells are , therefore , seen ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall