The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 43
SECTION XVI – PLATE 8 NORMAL ALKALINE PHOSPHATASE FROM BONE
NORMAL BLOOD LEVEL MARKED ELEVATION OF BLOOD LEVEL SO GO 20
OROSO OS 20 EXCRETION IN BILE DOBRO OS COMMON DUCT
OBSTRUCTION ...
SECTION XVI – PLATE 8 NORMAL ALKALINE PHOSPHATASE FROM BONE
NORMAL BLOOD LEVEL MARKED ELEVATION OF BLOOD LEVEL SO GO 20
OROSO OS 20 EXCRETION IN BILE DOBRO OS COMMON DUCT
OBSTRUCTION ...
Page 83
BILE INFARCT The effects of biliary obstruction upon the liver itself , best
observed in biopsy specimens , develop more rapidly in complete than in
incomplete obstruction . The change first to appear is the accumulation of bile
pigment in the ...
BILE INFARCT The effects of biliary obstruction upon the liver itself , best
observed in biopsy specimens , develop more rapidly in complete than in
incomplete obstruction . The change first to appear is the accumulation of bile
pigment in the ...
Page 127
If the obstruction persists , the lining epithelium atrophies and flattens , the folds
disappear and the wall becomes transformed into a fibrous scar tissue , with
gradual disappearance of the muscle bundles and obliteration of the distinction ...
If the obstruction persists , the lining epithelium atrophies and flattens , the folds
disappear and the wall becomes transformed into a fibrous scar tissue , with
gradual disappearance of the muscle bundles and obliteration of the distinction ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall