The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 71
In patients with a relaxed abdominal wall or in thin indi viduals with low
diaphragms , the liver may be palpable even in the absence of hepatic disease .
In patients with biliary cirrhosis ( see page 84 ) or fatty metamor phosis ( see
pages 79 ...
In patients with a relaxed abdominal wall or in thin indi viduals with low
diaphragms , the liver may be palpable even in the absence of hepatic disease .
In patients with biliary cirrhosis ( see page 84 ) or fatty metamor phosis ( see
pages 79 ...
Page 76
It has also been demonstrated that patients with advanced cirrhosis of the liver
have an abnormal elevation of ... of renal disease , and vice versa , one must be
prepared to treat the patient with hepatic disease who develops renal failure .
It has also been demonstrated that patients with advanced cirrhosis of the liver
have an abnormal elevation of ... of renal disease , and vice versa , one must be
prepared to treat the patient with hepatic disease who develops renal failure .
Page 138
Another group of patients with returning pains suffer from spasms of the sphincter
of Oddi , and rare cases have been seen with actual hypertrophy of the sphincter
responsible for a stenosis . Increasing formation of adhesions constricting the ...
Another group of patients with returning pains suffer from spasms of the sphincter
of Oddi , and rare cases have been seen with actual hypertrophy of the sphincter
responsible for a stenosis . Increasing formation of adhesions constricting the ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall