The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 16
... EDELSTEEDS PERB OPERATE SELE DE PRESENT CE E D Beth BEHEHEN
ES BE ANDEN SEBE , BERE ELLER The geograd DE D ELSE SEREBBE
EEEEEEEEEEEEE ENERGIE B ETTE ETE DERMED EUEELESENEGAL 359
ENERO ...
... EDELSTEEDS PERB OPERATE SELE DE PRESENT CE E D Beth BEHEHEN
ES BE ANDEN SEBE , BERE ELLER The geograd DE D ELSE SEREBBE
EEEEEEEEEEEEE ENERGIE B ETTE ETE DERMED EUEELESENEGAL 359
ENERO ...
Page 98
The liver cell plates are irregular , focal necroses are present and even
acidophilic round bodies may be seen . Most characteristic is the portal and
intralobular in filtration with round cells , some of which have indented nuclei .
These changes ...
The liver cell plates are irregular , focal necroses are present and even
acidophilic round bodies may be seen . Most characteristic is the portal and
intralobular in filtration with round cells , some of which have indented nuclei .
These changes ...
Page 113
Less frequent are other histologic varieties . The carcinoma cells may reveal
extensive fatty metamorphosis not present in the rest of the liver . Sometimes ,
large elongated multinuclear giant cells are present or even predominate the
picture .
Less frequent are other histologic varieties . The carcinoma cells may reveal
extensive fatty metamorphosis not present in the rest of the liver . Sometimes ,
large elongated multinuclear giant cells are present or even predominate the
picture .
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall