The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
From inside the book
Results 1-3 of 37
Page 62
Whatever the etiologic factors may be , the same textural picture of degeneration
is seen , though it should be kept in mind that more types may exist which cannot
be differentiated with the presently available morphologic techniques .
Whatever the etiologic factors may be , the same textural picture of degeneration
is seen , though it should be kept in mind that more types may exist which cannot
be differentiated with the presently available morphologic techniques .
Page 64
Changes of this kind are seen , for instance , in anemic infarcts ( see page 107 )
or as results of arterial diseases ( see page 109 ) or in areas with severed blood
supply following trauma ( see page 116 ) . Of greater significance is the necrosis
...
Changes of this kind are seen , for instance , in anemic infarcts ( see page 107 )
or as results of arterial diseases ( see page 109 ) or in areas with severed blood
supply following trauma ( see page 116 ) . Of greater significance is the necrosis
...
Page 94
... on epidemiologic evidence , that almost all cases of " acute yellow atrophy ” or
massive necrosis , at least as seen in Western Europe and North America , are
the result of the most severe phase of viral hepatitis , for which the term "
fulminant ...
... on epidemiologic evidence , that almost all cases of " acute yellow atrophy ” or
massive necrosis , at least as seen in Western Europe and North America , are
the result of the most severe phase of viral hepatitis , for which the term "
fulminant ...
What people are saying - Write a review
We haven't found any reviews in the usual places.
Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall