The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 11
It is , therefore , now assumed that the bile canaliculi have a fairly straight lining
with only small extensions between neighboring liver cells , a theory that agrees
with the picture seen in tissue sections if the bile capillaries are stained with ...
It is , therefore , now assumed that the bile canaliculi have a fairly straight lining
with only small extensions between neighboring liver cells , a theory that agrees
with the picture seen in tissue sections if the bile capillaries are stained with ...
Page 67
POSTNECROTIC CIRRHOSIS CHOLANGIOLITIC CIRRHOSIS HEPATIC TISSUE
COLLAPSES AFTER MASSIVE OR SUBMASSIVE NECROSIS OF AN AREA .
THIS LEADS TO STRESS IN SURROUNDING TISSUES , AND FISSURES ...
POSTNECROTIC CIRRHOSIS CHOLANGIOLITIC CIRRHOSIS HEPATIC TISSUE
COLLAPSES AFTER MASSIVE OR SUBMASSIVE NECROSIS OF AN AREA .
THIS LEADS TO STRESS IN SURROUNDING TISSUES , AND FISSURES ...
Page 141
N ES SECTION THROUGH NODULE OF PANCREATIC TISSUE IN GASTRIC
SUBMUCOSA BE OS DOBROD GOS segona NECROTIC ABSCESS ,
GANGRENE EXTENSIVE. SECTION XIX - PLATE I BRE 60 ANNULAR
PANCREAS ...
N ES SECTION THROUGH NODULE OF PANCREATIC TISSUE IN GASTRIC
SUBMUCOSA BE OS DOBROD GOS segona NECROTIC ABSCESS ,
GANGRENE EXTENSIVE. SECTION XIX - PLATE I BRE 60 ANNULAR
PANCREAS ...
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall