The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 10
CO Rose B RAS BE GE BEBE PER ER DOLORE ODO PORTAL TRIADS NET
biliary peritonitis by their spontaneous rupture or as a result of liver biopsy . Over
the normally smooth capsular convexity , fibrin may be deposited when plasma ...
CO Rose B RAS BE GE BEBE PER ER DOLORE ODO PORTAL TRIADS NET
biliary peritonitis by their spontaneous rupture or as a result of liver biopsy . Over
the normally smooth capsular convexity , fibrin may be deposited when plasma ...
Page 68
The septa , which have developed in different parts of the lobule , converge in
further stages of the process so as to create connections between portal triads
and central fields . These connections carry vessels which become anastomoses
.
The septa , which have developed in different parts of the lobule , converge in
further stages of the process so as to create connections between portal triads
and central fields . These connections carry vessels which become anastomoses
.
Page 92
ARTERIAL IEEE INFLAMMATORY FOCUS IN AND AROUND PORTAL TRIAD
HEPATIC ARTERY BRANCH PORTAL VEIN ... they are drained to the lymphatics
in the portal triads to set up an inflammation within and around the lymphatics .
ARTERIAL IEEE INFLAMMATORY FOCUS IN AND AROUND PORTAL TRIAD
HEPATIC ARTERY BRANCH PORTAL VEIN ... they are drained to the lymphatics
in the portal triads to set up an inflammation within and around the lymphatics .
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Contents
SECTION | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
15 other sections not shown
Common terms and phrases
abnormal abscesses acid acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture PLATE portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall