The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tractCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 126
... wall to a diffuse scarring with destruction of the epithelial lining and transformation of the wall into thick white scar tissue con- tracting the lumen . Although acute and chronic cholecystitis can cause stone for- mation ( see pages ...
... wall to a diffuse scarring with destruction of the epithelial lining and transformation of the wall into thick white scar tissue con- tracting the lumen . Although acute and chronic cholecystitis can cause stone for- mation ( see pages ...
Page 127
... wall becoming thin and stretched . In earlier stages the lining epithelium secretes an increased amount of mucus . If the obstruction per- sists , the lining epithelium atrophies and flattens , the folds disappear and the wall becomes ...
... wall becoming thin and stretched . In earlier stages the lining epithelium secretes an increased amount of mucus . If the obstruction per- sists , the lining epithelium atrophies and flattens , the folds disappear and the wall becomes ...
Page 130
... wall of the gall- bladder is often transformed into fibrous scar tissue , which , in smaller or larger areas , may be calcified . Such plaques are distinctly visible from the outside as well as after opening . The whitish , glisten- ing ...
... wall of the gall- bladder is often transformed into fibrous scar tissue , which , in smaller or larger areas , may be calcified . Such plaques are distinctly visible from the outside as well as after opening . The whitish , glisten- ing ...
Common terms and phrases
abdominal abnormal abscesses acid amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. CIBA nodes normal OCIBA organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall