The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
From inside the book
Results 1-3 of 49
Page 10
SUBLOBULAR VEIN CENTRAL VEIN CENTRAL VEIN ( Continued from page 9 )
TE 11 . di UN LIV M . 1 OUT FO V X V PORTAL TRIADS - biliary peritonitis by
their spontaneous rupture or as a result of liver biopsy . Over the normally smooth
...
SUBLOBULAR VEIN CENTRAL VEIN CENTRAL VEIN ( Continued from page 9 )
TE 11 . di UN LIV M . 1 OUT FO V X V PORTAL TRIADS - biliary peritonitis by
their spontaneous rupture or as a result of liver biopsy . Over the normally smooth
...
Page 12
OUTLET SPHINCTERS INLET SPHINCTERS ( Continued from page 11 )
CENTRAL VEIN 29 - OUTLET SPHINCTERS CENTRAL VEIN DISTRIBUTING
VEINS INLET VENULES WITH INLET SPHINCTERS PERIPHERAL SINUSOID
RADIAL ...
OUTLET SPHINCTERS INLET SPHINCTERS ( Continued from page 11 )
CENTRAL VEIN 29 - OUTLET SPHINCTERS CENTRAL VEIN DISTRIBUTING
VEINS INLET VENULES WITH INLET SPHINCTERS PERIPHERAL SINUSOID
RADIAL ...
Page 106
CENTRAL NECROSIS AND DISTENDED SINUSOIDS ACUTE STAGE V
DIAGRAMNECROSIS ( RED ) AROUND CENTRAL VEINS CARDIAC Liver "
BRIDGE FORMATION " BETWEEN CONGESTED CENTRAL ZONES
SUBACUTE STAGE ...
CENTRAL NECROSIS AND DISTENDED SINUSOIDS ACUTE STAGE V
DIAGRAMNECROSIS ( RED ) AROUND CENTRAL VEINS CARDIAC Liver "
BRIDGE FORMATION " BETWEEN CONGESTED CENTRAL ZONES
SUBACUTE STAGE ...
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall