The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 66
However , the formation of connective tissue membranes around the portal tracts
, as a result of inflammatory reactions partially secondary to liver cell injury , are
more important . These membranes extend from the tracts into the parenchyma ...
However , the formation of connective tissue membranes around the portal tracts
, as a result of inflammatory reactions partially secondary to liver cell injury , are
more important . These membranes extend from the tracts into the parenchyma ...
Page 78
The most distinctive feature is the diffuse increase of connective tissue
membranes throughout the parenchyma , with some septa transversing the
lobule to form portahepatic venous connections . Regenerative nodules , as a
rule , still remain ...
The most distinctive feature is the diffuse increase of connective tissue
membranes throughout the parenchyma , with some septa transversing the
lobule to form portahepatic venous connections . Regenerative nodules , as a
rule , still remain ...
Page 111
The smallest hamartomas , called microhamartomas or Meyenberg complexes ,
are irregular bile duct proliferations surrounded by proliferated connective tissue
( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture ...
The smallest hamartomas , called microhamartomas or Meyenberg complexes ,
are irregular bile duct proliferations surrounded by proliferated connective tissue
( see also page 60 ) . Solid , nodular hamartomas differ in color and architecture ...
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall