The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 25
A larger bud develops dorsal and proximal just above the level of the hepatic
diverticulum ( see page 2 ) . Growing fairly rapidly and extending into the dorsal
mesentery of the duodenum near the developing omental bursa , the dorsal ...
A larger bud develops dorsal and proximal just above the level of the hepatic
diverticulum ( see page 2 ) . Growing fairly rapidly and extending into the dorsal
mesentery of the duodenum near the developing omental bursa , the dorsal ...
Page 65
SEPTA DEVELOP IN THESE AS WELL , AS A RESULT OF LESS SEVERE
INVOLVEMENT OF SURROUNDING PARENCHYMA . REGENERATION
STARTS ( PURPLE ) PR GU EV es VO NO . Vetlara ROS CIBA IN LATER
STAGES , AS ...
SEPTA DEVELOP IN THESE AS WELL , AS A RESULT OF LESS SEVERE
INVOLVEMENT OF SURROUNDING PARENCHYMA . REGENERATION
STARTS ( PURPLE ) PR GU EV es VO NO . Vetlara ROS CIBA IN LATER
STAGES , AS ...
Page 105
In the snail ' s digestive gland the larvae pass through several stages ( sporocysts
) and develop into cercariae , which , having left the snail , propel themselves
with a forked tail . They are most active in shallow water exposed to sunlight ...
In the snail ' s digestive gland the larvae pass through several stages ( sporocysts
) and develop into cercariae , which , having left the snail , propel themselves
with a forked tail . They are most active in shallow water exposed to sunlight ...
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall