The Ciba Collection of Medical Illustrations: Digestive system: pt. 1. Upper digestive tract. [c1959]. pt. 2. Lower digestive tract. [c1962, 1979]. pt. 3. Liver, biliary tract, and pancreas. [2d ed., c1964 |
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Page 52
The total evacuation period of the gallbladder varies from 15 minutes to several
hours . The pattern of contraction exhibits great individual variation . The natural
stimuli for the release of bile into the duodenum ( cholecystokinetic effect ) , i . e .
The total evacuation period of the gallbladder varies from 15 minutes to several
hours . The pattern of contraction exhibits great individual variation . The natural
stimuli for the release of bile into the duodenum ( cholecystokinetic effect ) , i . e .
Page 68
... DAMAGE EFFECTS PORTAL HYPERTENSION EFFECTS HYPERESTRINISM
HYPERSPLENISM COMA JAUNDICE ... and normochromic type by
hypersplenism as well as probably by a nonspecific toxic effect upon the bone
marrow .
... DAMAGE EFFECTS PORTAL HYPERTENSION EFFECTS HYPERESTRINISM
HYPERSPLENISM COMA JAUNDICE ... and normochromic type by
hypersplenism as well as probably by a nonspecific toxic effect upon the bone
marrow .
Page 116
Internal stress or counter - coup effects during a blunt injury may set up
subcapsular or central lacerations or only a subcapsular ... a frequent
complication , has been associated with a toxic effect from tissue breakdown
products from the liver .
Internal stress or counter - coup effects during a blunt injury may set up
subcapsular or central lacerations or only a subcapsular ... a frequent
complication , has been associated with a toxic effect from tissue breakdown
products from the liver .
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Contents
SECTION XV | 1 |
PART I | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective contain continued cystic duct cysts cytoplasm damage depending develop diagnosis disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose hepatic artery increased indicates infection inferior injury instances intestinal intrahepatic involved iron jaundice later lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occurs organs origin pain pancreatic patients period pigment plates portal vein portion posterior present pressure primary produce protein rare reaction result seen serum severe sometimes space splenic stage stones structures superior surface surgical surrounding tests tion tissue tract tumor usually vary vessels viral wall